Our Cardiovascular Research Products in Action
Athens provides native human proteins that meet the demands of modern cardiovascular research, empowering teams to investigate physiological and pathophysiological processes, discover and validate biomarkers, develop diagnostics, and innovate therapeutics.
The Athens Advantage:
- Broad cardiovascular research product portfolio, spanning coagulation, cardiometabolic, vascular, and immune biology
- Native structure and activity, enabling target discovery and validation across diverse cardiovascular pathways
- High purity and reproducibility, ensuring confidence in signal stability and reducing lot-to-lot variability
- Consistent quality, form, fit, and function from mg to kg, simplifying revalidation as you scale
- High yields and batch reservations from single or pooled donors, supporting long-term cardiovascular research and manufacturing workflows
Explore how researchers worldwide have used Athens products to power cardiovascular research:
- Contois, et al. developed rapid ELISA assays to accurately measure Lipoprotein(a), an important cardiovascular risk factor1
- Mehta, et al. defined the role of hemopexin in atherosclerosis and vascular inflammation2
- Chen, et al. characterized heme-induced neutrophil extracellular traps (NETs) in sickle cell disease3
Want a deeper look?
Explore peer-reviewed studies featuring our cardiovascular research products.
*1. Contois, et al. Lipoprotein(a) particle number assay without error from apolipoprotein(a) size isoforms. Clinica Chimica Acta 505, 2020, 119–124. https://doi.org/10.1016/j.cca.2020.02.030 / Sun Diagnostics, USA
*2. Mehta, et al. Apolipoprotein E-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function. Arterioscler Thromb Vasc Biol. 2016 Jun;36(6):1152-63. https://doi.org/10.1161/ATVBAHA.115.306991
*3. Chen, et al. Heme-induced neutrophil extracellular traps contribute to the pathogenesis of sickle cell disease. Blood. 2014 Jun 12;123(24):3818-27. https://doi.org/10.1182/blood-2013-10-529982
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